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Mechanism of action and antimicrobial efficacy of synthetic compounds against drug-resistant gram-positive and gram-negative bacteria
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Mechanism of action and antimicrobial efficacy of synthetic compounds against drug-resistant gram-positive and gram-negative bacteria

Lumu Merdie Ntumba
Master of Science (MSc), University of Johannesburg
2025
Handle:
https://hdl.handle.net/10210/519061

Abstract

The increasing prevalence of multidrug-resistant bacteria presents a major public health challenge, necessitating novel antimicrobial strategies. This study evaluates the antimicrobial activity and mechanisms of action of three synthetic heterocyclic compounds: Compound A (2-amino-4-phenylpyrimido[1,2-a]benzimidazole), Compound B (2-imino-4-phenyl-2H-1,3- thiazino[3,2-a]benzimidazole), and Compound C (2-imino-4-phenyl-pyrimido[1,2- a]benzoxazole). Their antibacterial effects were assessed against Escherichia coli (Gramnegative), Staphylococcus aureus (Gram-positive), and Mycobacterium smegmatis (acid-fast) using microbiological and mechanistic assays. All three compounds exhibited an MIC of 15.625 μg/mL, with species-specific MBC variations. E. coli displayed a higher MBC for Compound B (62.5 μg/mL), while S. aureus and M. smegmatis had uniform MBCs of 31.25 μg/mL across all compounds. Mechanistic studies revealed that Compound B significantly increased bacterial membrane permeability (57.0% in S. aureus, 43.6% in E. coli), suggesting a membrane-destabilizing effect. Compound C exhibited the strongest efflux pump inhibition in E. coli (50.3% ethidium bromide efflux reduction at 32 μg/mL), comparable to verapamil (52.1% inhibition). Molecular docking studies predicted strong interactions between Compound C and multiple efflux-associated proteins, particularly in 8TTE (-8.3 kcal/mol) and 8QKK (-8.0 kcal/mol), supporting its potential as an efflux pump inhibitor. Compound A also exhibited high-affinity binding (-8.1 kcal/mol in 8TTE), reinforcing its inhibitory potential. In contrast, Compound B displayed moderate docking scores (-7.5 to -7.9 kcal/mol), with stable interactions in 4C48 involving key hydrophobic residues such as PHE727 and TRP809. However, experimental validation of direct target binding is necessary. Synergy analysis revealed that Compound B and Compound A exhibited strong synergy (FIC = 0.4) against S. aureus, while the triple combination (A+B+C) produced additive effects (FIC = 1.4) rather than enhanced synergy. Additionally, a non-linear fluorescence response was observed in M. smegmatis, suggesting potential fluorescence quenching or altered permeability dynamics at high concentrations. These findings highlight the potential of these compounds in antimicrobial development, particularly for targeting efflux-mediated resistance and bacterial membrane integrity. However, cytotoxicity testing was not conducted in this study, and future work will be necessary to evaluate the safety profile of these compounds in mammalian systems. Further studies are required to assess their cytotoxicity, antibiotic potentiation effects, and pharmacokinetics to determine their clinical applicability.
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